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Volume 16, Issue 1, January 2024

Reviews

The next decade of SET: from an oncoprotein to beyond
Han Yao, Meng Zhang, Donglai Wang
J Mol Cell Biol, Volume 16, Issue 1, January 2024, mjad082, https://doi.org/10.1093/jmcb/mjad082
Abstract | Full Text 

Articles

Pathologically relevant aldoses and environmental aldehydes cause cilium disassembly via formyl group-mediated mechanisms
Te Li, Min Liu, Fan Yu, Song Yang, Weiwen Bu, Kai Liu, Jia Yang, Hua Ni, Mulin Yang, Hanxiao Yin, Renjie Hong, Dengwen Li, Huijie Zhao, Jun Zhou
J Mol Cell Biol, Volume 16, Issue 1, January 2024, mjad079, https://doi.org/10.1093/jmcb/mjad079
Abstract | Full Text 
Depletion of Gsdma1/2/3 alleviates PMA-induced epidermal hyperplasia by inhibiting the EGFR–Stat3/Akt pathway
Qiyao Liu, Manyun Li, Minli Sun, Ruyue Xin, Yushu Wang, Qin Chen, Xiang Gao, Zhaoyu Lin
J Mol Cell Biol, Volume 16, Issue 1, January 2024, mjad080, https://doi.org/10.1093/jmcb/mjad080
Abstract | Full Text 
The structure of TRAF7 coiled-coil trimer provides insight into its function in zebrafish embryonic development
Xiaozhen Song, Ruixing Hu, Yi Chen, Man Xiao, Hong Zhang, Shengnan Wu, Qing Lu
J Mol Cell Biol, Volume 16, Issue 1, January 2024, mjad083, https://doi.org/10.1093/jmcb/mjad083
Abstract | Full Text 
Rbfox1 controls alternative splicing of focal adhesion genes in cardiac muscle cells
Peter Zorn, Jaime Calvo Sánchez, Tala Alakhras, Barbara Schreier, Michael Gekle, Stefan Hüttelmaier, Marcel Köhn
J Mol Cell Biol, Volume 16, Issue 1, January 2024, mjae003, https://doi.org/10.1093/jmcb/mjae003
Abstract | Full Text 

Letter to the Editor

Inducible RNA targeting and N6-methyladenosine editing by a split-Cas13 architecture
Yang Li, Qiang Sun, Zhi Yang, Gang Yuan
J Mol Cell Biol, Volume 16, Issue 1, January 2024, mjae002, https://doi.org/10.1093/jmcb/mjae002
Abstract | Full Text 

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Cover: The skin is like a wall to protect against injuries caused by external attacks. PMA represents the attack by the monster outside the wall. Upon PMA stimulation, GSDMAs act as sentinels to recruit EGFR ligands. The upregulated EGFR–Stat3/Akt signals thicken the wall to defend against the attack. In contrast, without GSDMAs, the response of the EGFR–Stat3/Akt pathway to external attack is decreased. See mjad080 by Liu et al. for details.